Melatonin Side Effects You Should Know

Published: 11th February 2011
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Feature of the melatonin is also the fact that the inversion of sleep ,it restores the disturbed biorhythm "sleep-wake. It was also noted significant reduction in the level of depression. Personal and reactive anxiety remained without dynamics. In neurologic picture of the dynamics were observed, which is apparently due to lack of time to detect a difference. Concluded that melaxen has a positive effect on sleep quality in the breach, caused by stroke.

But not only the improvement cycle "sleep-wake makes melatonin interesting for use in stroke patients. A number of studies (both experimental and clinical) Identify the most important properties of melatonin for the treatment of these patients:

1. melatonin increases cerebral reperfusion in rats with experimental arterial occlusion;
2. Melatonin reduces cerebral edema in rats with experimental stroke;
3. Melatonin enhances neuroplasticity in stress caused by experimental stroke;
4. with congenital hypoplasia of the pineal gland increases the risk of stroke and myocardial infarction;
5. changes in immune status in stroke may be related to impaired nocturnal secretion of melatonin;
6. Melatonin enhances neuroplasticity in the elderly.

Epilepsy. A sufficient number of studies show a decrease in nocturnal secretory activity of pineal gland in patients with epilepsy, while noting the lower levels of melatonin in patients with frequent attacks. Thus, as a result of these changes developing in the body of melatonin deficiency may be one reason for increased generation in the brain tissue of free radicals, which invariably accompanies the epileptic process. Prolonged use of anticonvulsants increases the formation of free radicals, which leads to oxidative stress and subsequent neuronal death. Increasing the concentration of free radicals in itself leads to the progression of the disease (degeneration of neurons as a result of lipid peroxidation and reduced glutathione in sinetza epileptic focus). In view of the above anti-stress and melatonin becomes clear the necessity of its use in these patients. Melatonin should be added to the basal anticonvulsant therapy and in conjunction with the presence of the neuroprotective properties as an inhibitor of glutamate receptors and the activator of the GABA receptors.

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